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Pain Perception in Mice Lacking the beta 3 Subunit of Voltage-activated Calcium Channels
Manabu Murakami et al.
Journal of Biological Chemistry 277 (43), 40342-51 (18 Oct 2002)
Posted by swensen1 to beta Calcium pain on Wed Jul 23 2008 at 16:08 UTC | info | related
 
Intracellular calcium dynamics at the core of endocardial stationary spiral waves in Langendorff-perfused rabbit hearts
Liang Tang et al.
AJP - Heart and Circulatory Physiology 295 (1), H297-304 (01 Jul 2008)
Posted by rclayton to Calcium on Tue Jul 15 2008 at 13:07 UTC | info | related
 
Similar effects of alpha- and beta-SNAP on Ca(2+)-regulated exocytosis.
A W Sudlow et al.
FEBS letters 393 (2-3), 185-8 (16 Sep 1996)
 
Role of calcium in the pathway for milk protein secretion and possible relevance for mammary gland physiology.
R D Burgoyne, J S Duncan, and A W Sudlow
Biochemical Society symposium 63, 91-100 (1998)
 
A hypo-osmotically induced increase in intracellular Ca2+ in lactating mouse mammary epithelial cells involving Ca2+ influx.
A W Sudlow and R D Burgoyne
Pflügers Archiv : European journal of physiology 433 (5), 609-16 (Mar 1997)
 
NMDA receptors amplify calcium influx into dendritic spines during associative pre- and postsynaptic activation
Jackie Schiller, Yitzhak Schiller, and David Clapham
Nat Neurosci 1 (2), 114-8 (Jun 1998)
This paper describe that supralinear summation of Calcium transient following Pre-post pairing stimulation of a spine is mainly contributed by increase of NMDA receptor Ca influx
 
Lithium desensitizes brain mitochondria to calcium, antagonizes permeability transition, and diminishes cytochrome C release.
Natalia Shalbuyeva, Tatiana Brustovetsky, and Nickolay Brustovetsky
The Journal of biological chemistry 282 (25), 18057-68 (22 Jun 2007)
 
Species- and tissue-specific relationships between mitochondrial permeability transition and generation of ROS in brain and liver mitochondria of rats and mice.
Alexander Panov et al.
American journal of physiology. Cell physiology 292 (2), C708-18 (Feb 2007)
 
Mitochondrial dysfunction is a primary event in glutamate neurotoxicity.
A F Schinder et al.
The Journal of neuroscience : the official journal of the Society for Neuroscience 16 (19), 6125-33 (01 Oct 1996)
 
Disease or no Disease?
bacteriality.com

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